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dc.contributor.authorKeynan, Y
dc.contributor.authorJuno, J
dc.contributor.authorMeyers, A
dc.contributor.authorBall, TB
dc.contributor.authorKumar, A
dc.contributor.authorRubinstein, E
dc.contributor.authorFowke, KR.
dc.date.accessioned2013-11-26T16:29:37Z
dc.date.available2013-11-26T16:29:37Z
dc.date.issued2010-10
dc.identifier.citationEmerg Infect Dis. 2010 Oct;16(10):1621-2. doi: 10.3201/eid1610.100108.en
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/pubmed/20875295
dc.identifier.urihttp://hdl.handle.net/11295/60619
dc.description.abstractBecause chemokine receptor 5 (CCR5) may have a role in pulmonary immune response, we explored whether patients with severe pandemic (H1N1) 2009 were more likely to carry the CCR5Δ32 allele than were members of the general population. We found a large proportion of heterozygosity for the CCR5Δ32 allele among white patients with severe disease.en
dc.language.isoenen
dc.publisherUniversity of Nairobien
dc.titleChemokine receptor 5 △32 allele in patients with severe pandemic (H1N1) 2009.en
dc.typeArticleen
local.publisherDepartment of Medical Microbiology,en


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