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dc.contributor.authorChoi Robert Y.
dc.contributor.authorFowke Keith R.
dc.contributor.authorJuno Jennifer
dc.contributor.authorLohman-Payne Barbara
dc.contributor.authorOyugi Julius O.
dc.contributor.authorBrown Elizabeth R.
dc.contributor.authorBosire Rose.
dc.contributor.authorJohn-Stewart Grace.
dc.contributor.authorFarquhar Carey.
dc.date.accessioned2013-02-28T06:24:40Z
dc.date.issued2012
dc.identifier.citationAIDS RESEARCH AND HUMAN RETROVIRUSES Volume 28, Number 6, 2012en
dc.identifier.urihttp://erepository.uonbi.ac.ke:8080/xmlui/handle/123456789/12200
dc.identifier.urihttp://online.liebertpub.com/doi/pdf/10.1089/aid.2011.0095
dc.description.abstractThe C868T single nucleotide polymorphism in the CD4 receptor encodes an amino acid substitution of tryptophan for arginine in the third domain. Previous studies suggest that C868T increases the risk of HIV-1 acquisition; however, the influence of this single nucleotide polymorphism (SNP) on disease progression has not been established. The presence of the C868T polymorphism was not statistically significantly associated with HIV-1 disease progression outcomes in a cohort of postpartum Kenyan womenen
dc.language.isoenen
dc.titleCc868t Single nucleotide polymorphism and HIV Type 1 Disease progression among postpartum women in Kenyaen
dc.typeArticleen


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