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dc.contributor.authorBlish, CA
dc.contributor.authorDogan, OC
dc.contributor.authorJaoko Walter G.
dc.contributor.authorMcClelland, RS
dc.contributor.authorMandaliya, K
dc.contributor.authorOdem-Davis, KS
dc.contributor.authorRichardsonb, BA
dc.contributor.authorOverbaugh, J
dc.date.accessioned2013-06-12T14:42:15Z
dc.date.available2013-06-12T14:42:15Z
dc.date.issued2012-03
dc.identifier.citationAIDS. 2012 Mar 13;26(5):643-6. doi: 10.1097/QAD.0b013e3283509a0b.en
dc.identifier.uriwww.ncbi.nlm.nih.gov/whalecom0/pubmed/22210637
dc.identifier.urihttp://erepository.uonbi.ac.ke:8080/xmlui/handle/123456789/32457
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC3511787/
dc.description.abstractA case-control study was performed to determine the effects of HIV-1-specific cellular immune responses on the odds of acquiring a second HIV-1 infection (superinfection). Changes in the frequency of cytokine-producing or cytolytic CD8+ or CD4+ T cells were not associated with significant alterations in the odds of superinfection, suggesting that HIV-1 specific cellular immune responses at the level induced by chronic infection do not appear to significantly contribute to protection from HIV-1 superinfection.en
dc.language.isoenen
dc.publisherUniversity of Nairobien
dc.titleCellular Immune Responses And Susceptibility To HIV-1 Superinfection: A Case-control Study.en
dc.typeArticleen
local.publisherDepartment of Infectious Diseasesen


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