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dc.contributor.authorIkumi, ML
dc.contributor.authorMuchohi, SN
dc.contributor.authorKokwaro, GO
dc.contributor.authorKokwaro, G
dc.contributor.authorNewton, CR
dc.date.accessioned2013-06-10T11:27:20Z
dc.date.available2013-06-10T11:27:20Z
dc.date.issued2008
dc.identifier.citationEpilepsy Res. 2008 Dec;82(2-3):215-8. doi: 10.1016/j.eplepsyres.2008.08.002. Epub 2008 Sep 19en
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/pubmed/18804958
dc.identifier.urihttp://erepository.uonbi.ac.ke:8080/xmlui/handle/123456789/30695
dc.description.abstractMalaria infection reduces the binding capacity of benzodiazepine receptors in mice. We studied the efficacy of diazepam terminating seizures in children with falciparum malaria. Diazepam stopped seizures in fewer patients with malaria parasitaemia (chi(2)=3.93, P=0.047) and those with clinical diagnosis of malaria (chi(2)=9.84, P=0.002) compared to those without. However malaria was not identified as an independent risk factor for diazepam's failure to stop seizures in children.en
dc.language.isoenen
dc.publisherUniversity of Nairobien
dc.titleResponse to diazepam in children with malaria-induced seizuresen
dc.typeArticleen
local.publisherCollege of Humanities Sciencesen


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